HIV: Against science
by Roberto A. Giraldo, Michael Ellner, Celia Farber, Barnett J. Weiss,
Francis R. Buianouckas, Tom DiFerdinando, and Edward A. Lieb
The treatment and prevention of AIDS with antiretroviral medications is based on a singular set of well known beliefs:
that AIDS is an infectious disease caused by a virulent virus called HIV; that HIV belongs to family of retroviruses; that
AIDS can therefore be treated with antiretroviral drugs; that AIDS is a transmissible disease that is transmitted through
body fluids including blood, genital secretions, and breast milk; that a positive result on the so-called "AIDS test"
is indicative of infection with HIV; that once positive on the "AIDS test" the individual will develop AIDS; that
a person who reacts positive on the "AIDS test" can prevent the development of AIDS by using several antiretroviral
drugs; that the consumption of antiretroviral drugs will prevent the transmission of HIV from HIV positive pregnant women
to their babies; that the use of antiretroviral drugs is safe and free of harmful effects; and that, therefore, it is rational
to treat and to prevent AIDS with antiretroviral medications.
However, not a single one of the above beliefs can be scientifically substantiated. On the contrary, there are many scientific
facts indicating that: the tests used for the diagnosis of HIV are extraordinarily inaccurate; that being HIV positive does
not mean that the person is infected with HIV, the so-called "AIDS virus"; that there are more than 70 different
non-HIV related reasons to have a positive result on the "AIDS test"; that the transmission and infectivity of AIDS
is not real; that the risk of developing AIDS after being labeled "HIV positive" is unknown; that HIV is not the
cause of AIDS; that HIV may not even exist as a virus; that what is called "AIDS" is a toxic and nutritional syndrome;
that all antiretroviral drugs are highly toxic to humans; that the antiretroviral medications can by themselves cause AIDS;
and that pregnant women, infants, and children are especially vulnerable to the toxic effects of antiretroviral medication.
It is not only irrational but indeed unethical to treat or prevent AIDS with toxic antiretroviral drugs in anybody. Besides
that, it is contrary to common sense to treat or prevent a highly toxicological syndrome with even more toxicity.
To treat or prevent AIDS with toxic antiretroviral medications is also a violation of the Universal Declaration of Human
Rights. Article 5 of the Universal Declaration of Human Rights states: "No one shall be subjected to torture or to cruel,
inhuman or degrading treatment or punishment." Therefore, no one has the right to "subject persons with HIV or AIDS
to inhuman and degrading treatment even if purportedly in the community's interests."
At the very least, there are serious legal implications with respect to the damage caused by these irrational treatments,
as well as possibilities for legal suits and monetary compensation.
It is likewise urgent that the entire conception of AIDS as an infectious and transmissible viral disease caused by HIV
be reappraised immediately.
People have the right to know both sides of a story, especially when they have to make decisions regarding their own health
care. Not informing people of all the facts - as mentioned in this article - is a serious violation of the person's right
to make informed consent medical decisions .
Self-determination and autonomy have been recognized, in fact, as a fundamental moral value in US law and are routinely
applied to a medical context. In the 1914 Schloendorff case, Justice Nenjamin Cordozo opined: Every Human Being of adult years
and sound mind has a right to determine what shall be done with his own body.
"The requirements for informed consent are as follows: 1) The practitioner must disclose all information, including
risks and benefits that a reasonable person would need to know in order to make a decision. 2) The one consenting must be
competent and must understand the information provided. 3) The consent must be given voluntarily and without coercion."
The full report, including footnotes, appears HERE
ABOUT THE AUTHORS:
Roberto A. Giraldo, Physician, Specialist in Infectious and Tropical Diseases. Member of the Board of Directors of the
Group for the Scientific Reappraisal of the HIV/AIDS Hypothesis and of Health Education AIDS Liaison, HEAL - New York.
Michael Ellner, Medical Hypnotherapist, Educator, Lecturer, President of HEAL - New York.
Celia Farber, Journalist. Member of the Board of Directors of the Group for the Scientific Reappraisal of the HIV/AIDHypothesis.
Barnett J. Weiss, CSW. Member of the Board of directors of HEAL - New York.
Francis R. Buianouckas, Ph.D. Professor of Mathematics. Member of the Board of Directors and Scientific Adviser of HEAL
- New York.
Tom DiFerdinando, Alternative pain and injury therapist, Educator, Lecturer, Executive Director of HEAL - New York.
Edward A. Lieb, Producer of Accent on Wellness. Owner of Planet Health. Member of the Board of Directors of HEAL - New
A Theory of Retrovirus
Viruses resident in the human genome may have evolved from jumping genes that increasingly appears to be necessary for the
survival of the organism. Some virologists think that we may be creating a range of debilitating chronic diseases by failing
to take them seriously into account. Dr. Mae-Wan Ho and Prof. Malcolm Hooper report to the Institute of Science in Society
Retroviruses are RNA viruses found in all vertebrates. The retrovirus reproduces by reverse transcribing (copying) its RNA
into DNA that is then integrated into the host-cell genome to be transcribed into viruses. Many retroviral sequences have
become permanently integrated into the human genome as human endogenous retroviruses, or HERVs. The human genome (indeed all
genomes) also contains retrovirus-like retrotransposons, mobile elements that multiply by making RNA copies that are reverse
transcribed into DNA and integrated into new sites in the genome. The main difference between a retrovirus and a retrotransposon
is that the latter lacks an envelope.
Most HERVs and retrotransposons are defective, having lost one or more gene functions; but can nevertheless multiply and move
with the help of other elements or infecting viruses.
Which came first, retrovirus or retrotransposon? As viruses depend on cells to reproduce, it seems reasonable to suppose that
retroviruses are simply retrotransposons that have gained an envelop, which helps in entering other cells. That idea was first
put forward by Howard Temin, who discovered reverse transcriptase, the enzyme that reverse transcribes, encoded by the retrovirus
Retrotransposons play an important role in turning genes on or off, and in rearranging and amplifying genes during development
and cell differentiation. And evidence has been accumulating that they are the natural molecular genetic engineers of the
fluid genome, which are necessary for the survival of the organism (see "Molecular genetic engineers in junk DNA?" this series).
HERVs are flanked by ‘long terminal repeats’ that contain strong promoters for gene expression. Promoters are
stretches of DNA with binding sites for transcription factors of the host cell that boosts transcription, effectively saying
to the cell, "make many copies of the message following". HERVs and retrotransposons are regulated by the cell, and ultimately,
by the organism as a whole, which stops most of them from being expressed; though they can be activated under a variety of
conditions (see below).
Many endogenous viruses show xenotropism, i.e., they are not active in the host cell but can become infectious to cells of
non-host species. Xenotropism is one of the major health hazards of xenotransplantation, the transplant of organs and tissues
between species, as from pig to humans.
Defective or dormant HERVs, like defective retrotransposons, can become expressed when missing gene-functions are provided
by a ‘helper’ virus that happens to infect the cell, and that includes ‘attenuated’ viruses in vaccines.
Like retrotransposons, HERVs can also be induced: by X-rays or various chemical agents and drugs, such as inhibitors of protein
synthesis, organophosphates and other pesticides, inflammatory cytokines (hormone-like factors that influence cells of the
immune system) or steroid hormones, and retinoic acid.
In a comprehensive review published in 1996, virologists Howard Urnovitz and William Murphy raised the possibility that many
chronic debilitating diseases may be linked to HERVs. These include leukaemia and other cancers, B-cell immunoglobulin diseases,
inflammatory diseases of the nervous system, autoimmune rheumatic and connective tissue disease and chronic fatigue syndrome.
There are several mechanisms linking HERVs with chronic diseases, though it is not at all clear which mechanism comes into
effect under different circumstances.
One way in which endogenous viruses can cause disease is for them to move and insert itself next to certain genes, that, when
over-expressed, results in uncontrolled cell division, or cancer. This mechanism may be involved in mouse and human leukaemia,
breast cancer and teratocarcinoma. This is also the mechanism that causes cancer in gene therapy, when viral vectors integrate
next to these same genes.
Another possibility is for an HERV to recombine with an infecting virus to generate new viruses. One theory for the origin
of the AIDS virus it that it may have come from early preparations of polioviruses used for vaccines that were propagated
in rhesus and African green monkey kidney cells. At least 26 monkey viruses, including adenoviruses, cosackievirus, herpesvirus,
echovirus, and possibly other groups of viruses were found as contaminants in such preparations. Current vaccines are presumably
free of such contamination, though that does not necessarily make them safe (see below).
Urnovitz and Murphy suggested that human immunodeficiency virus type 1 (HIV-1) may be a chimera between one of the simian
(monkey) viruses (simian immunodeficiency virus) and HERV sequences. Simian immunodeficiency virus capable of causing simian
AIDS, appears to occur exclusively in African monkeys, particularly in the African green monkey. And DNA sequences related
to the highly conserved domain of the HIV reverse transcriptase and glycoprotein gp41 (part of the gp160 polyprotein of the
HIV that’s cleaved into gp120 and gp41) have been found in the human genome.
Another way in which disease may arise is when HERV encoded proteins are expressed. This provokes antibodies against the body’s
own cells, giving rise to autoimmune diseases such as rheumatoid arthritis, lupus erythematosus, Sjögren’s syndrome,
mixed connective tissue diseases and inflammatory neurologic disease. The inflammatory response could be the most important
trigger for the development of autoimmune disease, as infecting viruses can strongly activate HERVs to express, resulting
in production of HERV protein antigens.
In this context, vaccines came in for special criticism in Unovitz’ testimony to the United States House of Representatives
In his view, "there appears to be a limit on how much foreign material to which the human body can be exposed before some
level of genetic damage occurs and a chronic disease initiates". Gulf War I veterans (GWIVs) were given large numbers of vaccines
(see "Gulf War Syndrome and vaccinations", this series), and vaccine overload is a significant factor in Gulf War Syndrome.
Urnovitz described a case of a woman who died from a mysterious case of AIDS. Over several years, laboratory tests failed
to reveal HIV-1 antibodies in her blood. However, she was subsequently tested HIV-1 positive in her urine. The virus was eventually
isolated and sequenced; and came to be known as HIV-1 Group O. Analyses of the viral genetic material suggest that the virus
originated, in part, from genetic reshuffling of human chromosomal material.
Vaccination against HIV-1 is particularly dangerous, if as Urnovitz and Murphy have suggested, HIV arose from recombination
between the simian aids virus and HERV sequences (see also "AIDS vaccines worse than useless?" this series).
"To put it simply, are we embarking on a course that will vaccinate people against their own genes?" Dr. Urnovitz asked.
A second example is the intensive effort directed to creating a vaccine for the hepatitis C virus. Unfortunately, there is
no hard evidence in the scientific literature that hepatitis C virus exists. Urnovitz continued, "As a scientist I am compelled
to ask, how can we vaccinate people against a disease-causing agent that has not been fully characterised?"
Finally, he drew attention to the contaminated polio vaccines that is now being increasingly implicated in cancer,
"Had my mother and father known that the poliovirus vaccines of the 1950s were heavily contaminated with more than 26 monkey
viruses, including the cancer virus SV40, I can say with certainty that they would not have allowed their children and themselves
to take those vaccines. Both of my parents might not have developed cancers suspected of being vaccine-related, and might
even be alive today."
But even uncontaminated Polio vaccines are of doubtful efficacy in protecting against viral infections and chronic disease.
Enteroviruses have been shown to be a major factor in Myalgic Encephalomyelitis/ Chronic Fatigue Syndrome by John Richardson
who studied more than 4000 patients over a 50 year period. Vaccination against polio provides protection against only 3 strains
of polio leaving no protection against the other 70 or so enteroviruses- coxsackie, echo and others. These viruses have a
range of pathological effects on the central nervous system, the cardiovascular system, and endocrine and exocrine glands.
The complete document with references, is available at http://www.i-sis.org.uk/ERCD.php
Crossreactivity of HIV Tests
Concerning the validity of HIV-tests. Its even more complicated-according
to Kashala et al. J. of Inf. diseases 1994;169,296-304 there is crossreactivity between HIV-1 and Antibodies to Lipoarabinomannan
which is found in Leprosy and Tuberculoses bacterias.So you will not know whether a patient is HIV+ because he has tuberculosis
or he has tuberculosis because he is HIV-pos. Treat Tuberculosis and don't care about HIV. I have done this for many years
and nobody died.
Email: Claus Khnlein:
The role of retroviruses in human life and disease
Human endogenous retroviruses (HERVs) have been the subject of a great deal
of investigation in recent years. It has been established that HERVs cause some
genetic diseases and there is strong circumstantial evidence for a link to
some types of cancer and autoimmune diseases, but what else do we need to know
about these unique organisms? In the December 2004 issue of the Journal of the Royal
Society of Medicine, Dr Frank Ryan discusses HERVs in the context of
medicine, human evolution and physiology.
Half of human DNA is viral
As Dr Ryan explains, endogenous retroviruses (ERVs) 'have the unique ability to invade the germ cells of every species of
vertebrate' where they are transmitted as part of normal reproduction (germline transmission), meanwhile interacting with
their host's evolution over millions of years. 'Genome sequencing reveals that 8% of the human genome consists of HERVs and
roughly half of our DNA is made up of viral genes or their genetic products.' So what are they really doing there?
Both useful and harmful to human life
HERVs also play a very important role in the normal human development and day to day existence. Three separate retroviruses
supply vital components to every human pregnancy. If, as the public might imagine, we should remove these viruses from our
chromosomes, the human species would become extinct. How do we reconcile these strange contradictions implicit in the behaviour
of the viruses?
HERVs and symbiosis
Symbiosis is the partnership of two different life forms. It does not imply a
friendly interaction. 'Certain microbes have entered into key symbiotic roles
with their hosts to enable a kind of fast-track evolution,' Dr Ryan says. 'We
breathe oxygen thanks to ancient bacteria that once invaded our distant
ancestor - the symbiotic descendants of these bacteria are the mitochondria in
every living cell today.' HERVs behave in much the same way. They begin as
'aggressive symbionts' - a complex form of parastism. But over time the relationship
evolves to a 'mutualistic symbiosis.' In symbioses, natural selection operates
not at the level of the selfish individual but at the level of the
partnership, honing each partner in such a way as to strengthen the viability of the
partnership, thus enabling it to become 'an evolutionarily stable strategy.'
There is growing evidence that this symbiotic interaction with very large numbers
of human retroviruses has played a major role in primate and most particularly
Understanding the broader perspective
Dr Ryan emphasises the importance of understanding the symbiotic nature of
HERVs to better understand their role in human diseases. 'It is not enough to
demonstrate a virus, or a viral product, in tissues affected by cancer or
disease. The viruses, or their products, may be playing a neutral or even a
beneficial role. We first need to understand such symbiotic roles before we can
recognise a pathological deviation that might contribute to a disease. This is
likely to become of increasingly important to our understanding of certain cancers and autoimmune diseases.'
Journal of the Royal Society of Medicine
As large as an elephant
Excerpt from a letter published in The British Medical Journal on-line edition:
It makes one tremble to think of the shame doctors will have to endure when the people of the world find out that they
had gotten it completely wrong about AIDS. The contagious, HIV hypothesis of AIDS is the biggest
scientific, medical blunder of the 20th Century. The evidence is as large as an elephant that AIDS is not contagious,
sexually transmitted, or caused by HIV. Shame is the main obstacle to exposing this simple fact. It is the fear of being so
obviously and hopelessly wrong about AIDS that keeps lips sealed, the money flowing and AIDS rhetoric spiraling to stratospheric
heights of absurdity.
The physicians who know or suspect the truth are embarrassed or afraid to admit that the HIV tests are absurd and should
be outlawed, and that the anti-HIV drugs are injuring and killing people. We are taught to fear antibodies, and to believe
that antibodies to HIV are a harbinger of disease and death ten years in the future. When you protest this absurdity and point
out to health care workers that antibodies are the very essence of anti-viral immunity your objections are met with either
contempt or embarrassed silence.
The only way we can free ourselves from the AIDS blunder and bring an end to the tyranny of fear is to have an open international
discourse and debate on all things AIDS. Anger will be a natural response to facing the enormity of the scandal of AIDS. Anger
has its place but it should be put aside quickly. It is a mistake to focus on villains and on whom to punish. The AIDS blunder
is a sociological phenomenon in which we all share a measure of responsibility.
The AIDS blunder shows that we need to rethink and restructure our institutions of government, science, health, academe,
journalism and media. We must replace the National Institutes of Health as the primary gatekeeper of research funding with
numerous competing sources of funding. We must restructure the peer review processes of scientific publishing and funding
so that they do not promote and protect any particular dogma or fashion of thought or exclude competing ideas. A robust and
mean investigative journalism must be revived, rewarded and cherished.
Visiting Scientist, Dept. Molecular & Cell Biology, UC Berkeley
NOTE: I respectfully disagree with Dr Rasnick - I think that we must focus on the villains and criminals like Faucci,
Gallo and Krim, et al.
As you know, I believe that most doctors, scientists and journalists were caught up in the hysteria and mass hypnosis
like every one else and should be viewed in that light.
However, since the publication of Duesberg's paper in 1987 - there have been more than a handful of scientists, doctors,
journal editors and journalists who in my opinion knowingly participated in covering up this murderous fraud and I believe
that we must hold them accountable.
The full letter to the editor can be found at:
Challenging the HIV theory
IN THE BRITISH MEDICAL JOURNAL
I must say that I was delighted and shocked to see that the following letters were published in the British Medical Journal (BMJ).
Based on my review of the collective works of the Perth Group, Stefan Lanka and Roberto Giraldo - "HIV" tests are non-specific! All positive "HIV results" are false positives!
HIV DOES NOT CAUSE AIDS
30 January 2002
TO THE EDITOR:
The AIDS establishments have spent the last twenty years focusing on the HIV and not on the real causes of AIDS. The correct approach for investigating the cause(s) of a disease is by evaluating all medical evidence that considers infectious, chemical, nutritional, and metabolic factors. As a pathologist and a toxicologist, I evaluated the published literature on the worldwide AIDS epidemic and found that HIV does not cause AIDS. In my book 'Get All the Facts: HIV Does Not Cause AIDS,' I described the multifactorial causes of AIDS in the world and explained the pathogenesis of AIDS in different risk groups [Mohammed Ali Al-Bayati, 'Get All The Facts: HIV does not cause AIDS' Toxi-Health International, Dixon CA 1999, 183 pages ISBN 0-9673536-0-2].
My findings include:
1) The HIV-hypothesis is not supported. HIV is a harmless virus in both the in vivo and the in vitro settings.
2) AIDS in drug users and homosexuals in the U.S. and Europe is actually caused by the heavy ancillary use of glucocorticoids and other immunosuppressive agents to medically treat the wide range of chronic serious illnesses of the respiratory system, gastrointestinal system, and other organs, malnutrition, release of endogenous cortisol, and opportunistic infections in these persons. The appearance of "AIDS" in the U.S. and Europe has coincided with the approval of glucocorticoid aerosoll use in 1976, the introduction of crack cocaine, the use of heroin by inhalation, and the use of alkyl nitrites by homosexuals to enhance sexual activities.
3) AIDS in hemophiliacs is related to the use of corticosteroids and other immunosuppressive agents to prevent the development of antibodies for factors VIII and IX and to treat other chronic illnesses such as joint disease.
4) AIDS in people receiving blood and/or tissue is related to the use of glucocorticoids to prevent reactions of transfusion and tissue rejection, and to treat other illnesses.
5) AIDS in infants and children is caused by their exposure to drugs and corticosteroids in utero and their exposure to corticosteroids used after birth to treat their chronic illnesses.
6) AIDS in Africa is caused by malnutrition, release of endogenous cortisol, and opportunistic diseases. Atrophy in the lymphoid tissue in people suffering from malnutrition has been known since 1925. Malnutrition causes severe atrophy in the thymus and lymphoid organs and impairs the function of the T cells. These changes are reversible by feeding. The size of the thymus in malnourished children increased from 20% of normal to 107% of normal, following nine weeks of feeding.
7) Kaposi's sarcoma (KS) and lymphoma are induced by the use of steroids and drugs, and the release of endogenous cortisol. They are not caused by a slow virus. KS is reversible upon the termination of treatment with immunosuppressive agents prior to metastasis.
8) The medications currently used to treat patients with AIDS, such as AZT, protease inhibitors, and glucocorticoids, are highly toxic. They can even cause AIDS in asymptomatic patients, and make the disease worse in patients with AIDS. These drugs do not have any therapeutic value, and their use must be discontinued immediately.
9) Damage to the immune system is rapidly reversible after removal of the true insulting agent or treatment of the true causes. For example. a) The CD4+ T cells of 1075 HIV+ pregnant women increased from 426/uL to 596/uL in six months by giving these women a balanced diet. This also improved the outcome of their pregnancy; and b) The reduction in CD4+ T cells in HIV homosexuals was also reversed by the cessation of treatment with glucocorticoids.
My findings have been reviewed and supported by scientists and physicians.
Mohammed Ali Al-Bayati,
President, Toxi-Health International
Toxi-Health International, 150 Bloom Dr., Dixon, CA 95620
Send response to journal:
Re: HIV Does Not Cause AIDS
Email Mohammed Ali Al-Bayati:
Below are the links to some of these articles and reviews: